E-mailed the first one but I don't have access to second one, sorry...

Title : Evidence for an immune response in major depression: a review and hypothesis
Authors : Michael Maes
Journal : Progress in Neuro-Psychopharmacology and Biological Psychiatry
Publication Date : 1995
Direct Link : "www.sciencedirect.com/science/article/pii/027858469400101M"
Abstract :
1. This paper reviews recent findings on cellular and humoral immunity and inflammatory markers in depression.
2. It is shown that major depression may be accompanied by systemic immune activation or an inflammatory response with involvement of phagocytic (monocytes, neutrophils) cells, T cell activation, B cell proliferation, an “acute” phase response with increased plasma levels of positive and decreased levels of negative acute phase proteins, higher autoantibody (antinuclear, antiphospholipid) titers, increased prostaglandin secretion, disorders in exopeptidase enzymes, such as dipeptidyl peptidase IV, and increased production of interleukin (IL)-1β and IL-6 by peripheral blood mononuclear cells.
3. It is hypothesized that increased monocytic production of interleukins (Il-1β and Il-6) in severe depression may constitute key phenomena underlying the various aspects of the immune and “acute” phase response, while contributing to hypothalamic-pituitary-adrenalaxis hyperactivity, disorders in serotonin metabolism, and to the vegetative symptoms (i.e. the sickness behavior) of severe depression.

Sent to your e-mail...

Still noone ?

Günaydınlar ve teşekkürler :)
Appreciating...

No access for anyone ?

I'm searching for a complete but effordable solution for small laboratory animal (rat, mice etc.) electroretinography. Do you have suggestions ?

My deepest thanks for all...

"...unpleasantness if you disagree with your supervisor" is the foremost issue ;)

Title : Characterization of serotonin neurotransmission in knockout mice: implications for major depression.
Authors : Sergio Domínguez-López, Rebecca Howell, Gabriella Gobbi
Journal : Reviews in the Neurosciences
Publication Date : 2012
Direct Link : "www.degruyter.com/view/j/revneuro.2012.23.issue-4/revneuro-2012-0044/revneuro-2012-0044.xml"
Abstract :
The interaction between genes and environment plays a significant role in the pathogenesis of major depression and mood disorders. Preclinical and clinical studies have established that a dysfunction of serotonin (5-HT) neurotransmission is a common hallmark in major depression and drugs acting on the 5-HT system have antidepressant properties. In the past 15 years, the development of knockout mice showing a depressive-like or resilience-like phenotype have allowed us to better understand the complex relationship between genes, behaviour and the 5-HT system in mood disorders. The present review revises several knockout mice genotypes with ‘mood’ alteration and analyses how 5-HT firing activity, measured with electrophysiological techniques, is impaired after a gene manipulation. The behavior and electrophysiology data from 5-HT transporter (5HTT), 5-HT1A, 5-HT4, the neurokinin 1 (NK1) receptor, fatty acid amide hydrolase (FAAH) and the TWIK-1 related K+ (TREK-1) channel knockout mice are here analysed. Interestingly, a correlation between 5-HT firing rate and depressive/resilience phenotypes can be established in these different knockouts. Furthermore, findings in knockout mice have been successfully translated to humans, and findings from human studies have helped to design and generate knockout mice to explore new hypotheses of the etiology of human depression. The correlation of 5-HT activity and behavior could be a predictor factor for understanding the role of receptors, channels and enzymes in depression, and could be used also to assess the potential antidepressive effects of novel drugs.

E-mailed first 2 pages. Hope it helps...

Title : Monoamine oxidase inhibitory components from Cayratia japonica.
Authors : Han, Xiang Hua; Hong, Seong Su; Hwang, Ji Sang; Lee, Myung Koo; Hwang, Bang Yeon; Ro, Jai Seup
Journal : Archives of Pharmacal Research
Publication Date : 2007
Direct Link : "link.springer.com/article/10.1007%2FBF02977772"
Abstract :
Seven flavonoids were isolated from the whole plants and fruits ofCayratia japonica through the activity-guided isolation of a methanol extract using a monoamine oxidase (MAO) inhibition assay as a monitor. The chemical structures of the isolates were assigned as apigenin-7-O-β-D-glucuronopyranoside (1), apigenin (2), luteolin (3), luteolin-7-O-β-D-glucopyranoside (4), (+)-dihydroquercetin (taxifolin) (5), (+)-dihydrokaempferol (aromadendrin) (6) and quercetin (7). Among the isolated compounds, flavones such as apigenin (2) and luteolin (3), as well as the flavonol, quercetin (7) showed potent inhibitory effects against the MAO activity with IC50 values of 6.5, 22.6, and 31.6 μM, respectively. However, the flavone glycosides, apigenin-7-O-β-D-glucuronopyranoside (1) and luteolin-7-O-β-D-glucopyranoside (4), showed mild MAO inhibition (IC50 values: 81.7 and 118.6 μM, respectively). The flavanonol derivatives, taxifolin (5) and aromadendrin (6), also showed weak inhibition (IC50 values: 154.7 and 153.1 μM, respectively). Furthermore, quercetin (7) had a more potent inhibitory effect on MAO-A (IC60 value: 2.8 μM) than MAO-B (IC50 value: 90.0 μ.M). Apigenin (2) and luteolin (3) also preferentially inhibited MAO-A (IC50 values: 1.7 and 4.9 μM, respectively) compared with MAO-B (IC50 values: 12.8 and 59.7 μM, respectively).

My suggestion is "ithenticate"... Institutions that have subscription mostly gives authorization to heads of departments. You may ask to head for checking.
... and If I were you, I would never take the risk of distribution. Not me but people store their project files as encrypted even in dropbox

*bump!*

Actually I don't have institutional access to Nature publ. but this article seems free one.
Whatever... Here it is :
"www.sendspace.com/file/1aeii4"

... and for deletion after download :
"www.sendspace.com/delete/1aeii4/7b67f9988e3d59e92ad4dfe8a9b82f09"